Given the recent failure of the Bapi clinical trial, there is a lot of questions that have arised from he amyloid hypothesis. However, I can't really think of many other mechanisms that don't involved plaque formation via protein aggregation.
What are the other dominant proposed mechanisms for the biochemistry of Alzheimers?
Reference: Alzheimer’s Drug Fails Its First Big Clinical Trial. Unfortunately, the results of the study have yet to be presented at a medical conference but will be in September.
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